What is the ketogenic diet?
The ketogenic diet is a high-fat, low-carbohydrate eating plan that shifts your body into ketosis — a metabolic state where fat, rather than glucose, becomes your primary fuel source. In practice, it means keeping total carbohydrate intake low enough (usually under 30–50 grams per day) that the liver begins producing ketone bodies from fat.
For beginners, success on keto means focusing on whole foods — meat, fish, eggs, dairy, non-starchy vegetables, nuts — and limiting grains, sugars, starches and most fruit. The target proportions are approximately 70–75% of calories from fat, 20–25% from protein, and 5–10% from carbohydrates.
The three macronutrient targets
A standard ketogenic day, expressed as a percentage of total calories:
| Macronutrient | % of calories | Grams on a 2 000 kcal day | Role |
|---|---|---|---|
| Fat | 70–75% | 155–167 g | Primary fuel source in ketosis. |
| Protein | 20–25% | 100–125 g | Preserves lean mass; adequate, not high. |
| Carbohydrate | 5–10% | 25–50 g | Kept low enough to sustain ketosis. |
What ketosis actually means
When dietary carbohydrates fall below roughly 50 g per day for several days, liver glycogen stores deplete. The liver then begins converting fatty acids into three ketone bodies — beta-hydroxybutyrate (BHB), acetoacetate, and acetone — which most tissues, including the brain, can use for energy.
Nutritional ketosis is conventionally defined as blood BHB concentrations between 0.5 and 3.0 mmol/L. This is distinct from diabetic ketoacidosis, a pathological state that occurs when insulin is absent — at ketone levels ten to twenty times higher.
Benefits and risks, with the evidence labelled
The ketogenic diet is well-studied in some contexts (epilepsy, short-term weight loss, type 2 diabetes) and under-studied in others (long-term cardiovascular outcomes, athletic performance). We cite a medical or academic source for the claims we make.
✓ Likely benefits
- Weight lossMeta-analyses show faster short-term loss vs. low-fat diets, largely through appetite reduction. Evidence: strong.
- Blood-sugar controlLower HbA1c and reduced medication in type 2 diabetes (Virta trial, 2-year data). Evidence: strong.
- Reduced hungerConsistently reported across trials; likely mediated by higher satiety from fat and protein. Evidence: moderate.
- Therapeutic use in epilepsyA first-line treatment for drug-resistant childhood epilepsy since the 1920s. Evidence: strong (clinical).
- Improved triglycerides / HDLLower triglycerides and higher HDL commonly reported. LDL response is individual. Evidence: moderate.
! Potential risks
- Keto flu (first 1–2 weeks)Headache, fatigue, irritability from electrolyte loss. Managed with salt, magnesium, potassium. Common, short-term.
- Raised LDL cholesterolA subset of lean individuals show large LDL increases. Monitor with lipid panel and ApoB. Variable.
- Nutrient gapsFibre, potassium, magnesium and folate intake can fall if non-starchy vegetables are neglected. Avoidable.
- Kidney stonesSmall increased risk in clinical-ketogenic protocols, particularly in children. Adequate hydration reduces risk. Uncommon.
- ContraindicationsPregnancy, type 1 diabetes, pancreatitis, certain genetic disorders. Always consult a clinician. Specific populations.
Foods to eat, and foods to avoid
The working principle is simple: prefer whole foods that occur in nature without a label, limit processed carbohydrates, and keep total carb intake within your target.
✓ Eat regularly
✗ Limit or avoid
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